Hello Ranchers, Industry et al,
A kind greetings from hot Bacliff, Texas.
As a layperson, and as a consumer, I signed on to this site to lurk, and see what was being said about BSE, TSE, the prion disease, aka mad cow disease. however, after i did a search with several TSE BSE prion related search terms, to my surprise, there were no hits...none. maybe there was an error in the search system here. so, i thought i could not just lurk here. but please let me try and explain here what my intentions are, before the shinola hits the fan, and everybody tries to shoot the messenger. believe, me, i been shot before.
i am not here as an anti-meat, anti-hunter, advocate. i advocate nothing but the truth. i am a meat eater. i am a carnivore. i have no PhDs either. i am here as strictly a layperson, but i am a consumer of your products as well. i just don't want TSE prions, E-coli, MRSA, feces, urine, antibiotics, hormones, ammonia, mystery meat, or any other dangerous pathogens and or chemicals on what i eat. i don't want to have to either eradiate or burn my meat. should not have to. i want to know where it comes from, where it's been, and i want it tested for prions, and as cheap as that would be, why has it not been done long ago? i want a NAIS Traceability put in place, from farm to fork.
I bet your ready to shoot the messenger/consumer already. please don't. please allow me to explain from a consumer point of view, one that has followed the research of the TSE prion disease in all species, day by day, year by year, for over 14 years now. followed MRSA/VRSA for about 10 years. I lost my Mother to the Heidenhain Variant of Creutzfeldt Jakob Disease 12/14/97, hvCJD an exceedingly rare strain of the sporadic CJD's, and please note, atypical BSE has now been linked to some sporadic CJD cases in North America. Also, i about died in 2001 from MRSA after a surgery. i have set up a series of data banks to store this data, and research there from. i do not make money, i do not advertise, there. i think science and studies there from, especially when much of the work is funded by the public, should be free to the public, especially from the ones that are looking for answers, and when the Government and Industry work so hard to keep it all from the public, consumer, domain.
i only hope that some sort of dialog, discussion, debate, can be brought forth here. one that is not hostile. but either way, i hope some, if not all here find this information useful and helpful.
I will post a few links, one to an article that ran in the Galveston Daily News recently, about a shipment of cattle to Russia, and where these cattle came from, and why TEXAS, ALABAMA, AND WASHINGTON, did not ship any cattle to Russia. they call it MAD COW DISEASE. nope you don't like to hear it, you want speak of it, but i am here to tell you, it's still here, it has not gone anywhere, and in fact, TSE prion disease is spreading in North America, in animals and humans. The BSE surveillance system, testing, and the infamous mad cow feed ban that never was, what they call the 'TRIPLE FIRE WALL', was terribly flawed at all fronts, and the OIE and the USDA are the ones at fault here. all this is fact. i wish to simply bring this information to your attention. i don't blame the farmers and ranchers, it's the government and the industry that has led the astray (in most cases. you still have a few that simply don't care).
look, everybody has an opinion, and that's their right, and i respect everyone's opinion. but with no discussion, there can be no opinions, and this disease is not going anywhere. so let's at least discuss it. ...
Good Luck !
kindest regards, terry
Wednesday, March 31, 2010
Atypical BSE in Cattle
To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE.
In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.
This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle.
Sunday, May 01, 2011
STUDY OF ATYPICAL BSE 2010 Annual Report May 2011
What is the potential cost of pre- and post-slaughter testing? The estimated cost of post-mortem testing is $40 per head. This amount is comprised almost entirely of the cost of the test kit and sample analysis. It is expected that ante-mortem tests (live animal), if a test is developed, will reduce BSE testing costs to approximately $15 per head.
Wednesday, June 15, 2011
Galveston, Texas - Isle port moves through thousands of heifers headed to Russia, none from Texas, Alabama, or Washington, due to BSE risk factor
Please see the following warning from CDC about prion TSE consumption in North America ;
Thursday, May 26, 2011
Travel History, Hunting, and Venison Consumption Related to Prion Disease Exposure, 2006-2007 FoodNet Population Survey
Journal of the American Dietetic Association Volume 111, Issue 6 , Pages 858-863, June 2011.
Thursday, June 2, 2011
USDA scrapie report for April 2011 NEW ATYPICAL NOR-98 SCRAPIE CASES Pennsylvania AND California
Thursday, August 12, 2010
Seven main threats for the future linked to prions
The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.
***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.
Rural and Regional Affairs and Transport References Committee
The possible impacts and consequences for public health, trade and agriculture of the Government’s decision to relax import restrictions on beef Final report June 2010
2.66 Dr Fahey also told the committee that in the last two years a link has been established between forms of atypical CJD and atypical BSE. Dr Fahey said that: They now believe that those atypical BSEs overseas are in fact causing sporadic Creutzfeldt-Jakob disease. They were not sure if it was due to mad sheep disease or a different form. If you look in the textbooks it looks like this is just arising by itself. But in my research I have a summary of a document which states that there has never been any proof that sporadic Creutzfeldt-Jakob disease has arisen de novo—has arisen of itself. There is no proof of that. The recent research is that in fact it is due to atypical forms of mad cow disease which have been found across Europe, have been found in America and have been found in Asia. These atypical forms of mad cow disease typically have even longer incubation periods than the classical mad cow disease.50
Tuesday, June 14, 2011
Clinical research in CJD at a U.S. clinical prion research center: CJD Quinacrine Study results and improved diagnosis of prion disease
now you can shoot the messenger. ...
kindest regards, terry